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Four found energy intake to be significantly reduced, whereas the remaining studies failed to find an effect again potentially due to limited statistical power and also low GLP-1 infusion rates

Traditionally, EECs are thought to release CCK through GPR40 activation, which induces cellular depolarization through intracellular calcium store release by inositol triphosphatedependent mechanisms 17

Dis Esophagus 27(2):168-75, 2014